La cocaína es un estimulante sumamente adictivo que afecta directamente al sistema nervioso central. Las principales vías de administración de la cocaína son oral, nasal, intravenosa y pulmonar. "Crack" es el polvo de clorhidrato de cocaína que ha sido procesado para hacer una roca de cristal que genera vapores que se fuman.
Nombres de la calle
En español se conoce como "coca", "nieve", "dama blanca" o "talco". Algunos de sus nombres en inglés son "coke", "C", "snow", "flake" y "blow".
A landmark study in mice identifies a biological mechanism that could help explain how tobacco products could act as gateway drugs, increasing a person’s future likelihood of abusing cocaine and perhaps other drugs as well, according to the National Institute on Drug Abuse (NIDA), part of the Nat
Las drogas estimulantes como la cocaína, el "crack", las anfetaminas y la cafeína son sustancias que aceleran la actividad del cerebro y de la médula espinal. Con frecuencia, los estimulantes ejercen influencia sobre el usuario de manera que se vuelve más hablador y ansioso o tiene una sensación de gran alegría.
The first in a 5-part series, offers an understanding of the brain, how the reward center works, and what happens in the brain when a person uses cocaine, opiates (heroine), or marijuana.
Describes an inpatient study investigating the effectiveness of sustained-release methylphenidate as a treatment for cocaine-addiction patients with ADHD.
Summarizes an animal study of the neural changes in the amygdala portion of the brain that occur as a result of cocaine exposure followed by withdrawal of the drug.
Discusses the work of NIDA’s Division of Basic Neuroscience and Behavioral Research, the Institute’s locus for studies into the fundamental brain mechanisms underlying drug abuse and addiction.
Reports on a study that documenting changing emotional and neurobiological responses to cocaine with successive doses during a single session of drug taking.
Reports on the work of researchers who used brain images to correlate cocaine-addicted patients' regional brain responses to drug cues with their outcomes in subsequent treatment.
